The Journal of Experimental Medicine
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Index of Online Supplemental Material for
J. Exp. Med. 10.1084/jem.20081370
Cavassani et al.

Figure S1 mRNA expression of TLR4 and 9 is increased in tlr3−/− mice after CLP.

Figure S2 TLR3 activation contributes to the injury induced by septic peritonitis and gut ischemia.

Figure S3 Vimentin protein and rHMGB1 are not involved in the inflammatory response triggered by the TLR3 pathway.

Figure S4 Anti-TLR3 antibody attenuated poly(I:C)-induced chemokine generation by WT macrophages but did not have any effect on chemokine production induced by TLR4, 9, and 2 ligands

Figure S5 The functional activity of neutralizing anti-TLR3 for inflammatory chemokine production is not dependent on endosomal acidification.





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