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J. Exp. Med. 10.1084/jem.20072327
Guo et al.

• Figure S1 Expression of early developmental markers in fresh splenic p110δ^D910A/D910A NK cells.

• Figure S2 IL-2–activated NK cells from p110δ^D910A/D910A mice express normal levels of developmental or activation markers.

• Figure S3 Catalytic inactivation of p110δ does not affect the expression of self–MHC class I molecules.

• Figure S4 Cell death is not the cause of reduced numbers of Ly49C/I subsets in p110δ^D910A/D910A mice.

• Figure S5 Cytotoxicity of NK cells against EL4^H60 is primarily mediated by NKG2D receptor.

• Figure S6 NK cells from p110δ^D910A/D910A mice have a decreased ability to clear tumor cells in vivo.

• Figure S7 Ability of IL-2–activated, BM-derived NK cells from p110δ^D910A/D910A mice to generate cytokines or chemokines is severely impaired.

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