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Index of Online Supplemental Material for
J. Exp. Med. 10.1084/jem.20070204
Narni-Mancinelli et al.

• Figure S1 Frequencies of CD8⁺ T_EM and CD8⁺ T_CM cells after secA2², actA², and WT bacteria infection.

• Figure S2 CCL3 from CD8⁺ T cells is required for secondary protective immunity in the liver.

• Figure S3 Kinetics of mice survival. BALB/c (A) or 129 control or p47^phox2/2 (B) mice (five per group) were injected with PBS or immunized with 3 3 10³ WT bacteria.

• Figure S4 CCL3 from CD8⁺ T cells is required for secondary protective immunity.

• Figure S5 The protection achieved by transferring WT CD8⁺ T cells increases when recipient animals are challenged with lower numbers of bacteria.

• Figure S6 Phenotypic characterization of TP-MPCs. BALB/c (three to four per group) primary immunized with 3 3 10³ WT bacteria were challenged with either PBS (A) or 3 3 10⁵ WT bacteria (A-D) and killed 10 h later.

• Figure S7 Frequencies of TP-MPCs in spleen and among total MPCs.

• Figure S8 Numbers of ROI-producing MPCs and neutrophils in primary and secondary infected mice.

• Figure S9 CCL3-producing memory CD8⁺ T cells promote ROI generation by phagocytic cells for secondary bacterial clearance.

• Figure S10 Number of granzyme B-expressing and IFN-g-secreting CD8⁺ T cells in WT and p47phox^2/2 129 mice after primary and secondary infection.

• Figure S11 ROI are required for mediating secondary protection against L. monocytogenes infection.

• Figure S12 Analysis of lysozyme M expression in MPCs.

 

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