ARTICLE

¹ From the Pathological Laboratory, Mount Sinai Hospital, New York.

It must be assumed that some cause is operative in certain cases preventing the healing of defects in the gastric mucosa and is inoperative in others. Even though anhemolytic streptococci are present in practically all gastric ulcers, we cannot convince ourselves that these organisms have been proven as yet to be the factor which either initiates the ulceration or prevents healing. Nevertheless, the constant presence of streptococci in this type of lesion is a suggestive fact and further experiments to determine their significance are being undertaken.

CiteULike    Complore    Connotea    Del.icio.us    Digg    Facebook    Reddit    Technorati    Twitter    What's this?

This article has been cited by other articles:

• VONDERAHE, A. R. (1939). HISTOPATHOLOGIC CHANGES IN THE NERVOUS SYSTEM IN CASES OF PEPTIC ULCER. Arch Neurol Psychiatry 41: 871-912 [Abstract]  
• SAUNDERS, E. W. (1930). THE SEROLOGIC AND ETIOLOGIC SPECIFICITY OF THE ALPHA STREPTOCOCCUS OF GASTRIC ULCER: A BACTERIOLOGIC STUDY. Arch Intern Med 45: 347-382 [Abstract]  
• McCANN, J. C. (1929). EXPERIMENTAL PEPTIC ULCER. Arch Surg 19: 600-659 [Abstract]  
• KARSNER, H. T. (1925). THE PATHOLOGY OF PEPTIC ULCER OF THE STOMACH. JAMA 85: 1376-1380 [Abstract]  

Home | Help | Feedback | Subscriptions | Archive | Search

TABLE OF CONTENTS